Buffalo Bills Fan

Might be new noemal now. Sorry everyone. But in unite most counries need to blame China for there *****.People can call me out but I think might be new normal on how to live things. ***** china. President Trump, democrats, Republicans whatever everyone in this world needs to blame them.

Agree man

Agree GG more lethal viral load and horrible management by placing sick people into nursing homes. Plus we have better med, info, data to help people out (all of that).

Sorry this is just odd.

That's pretty nice. Happy 4th of July everyone.

Trump 'all for masks,' looked like 'Lone Ranger' in one President unsure if mandatory mask mandate is necessary Facebook Twitter Comments Print Email By Paul BestFOXBusiness Trump: I’ll wear a mask if I’m in a crowd President Trump, in an exclusive interview with FOX Business' Blake Burman, says he’s not against masks and does wear a mask when he’s close to a group of people. President Trump has been criticized by politicians for not wearing a mask in public, but he told FOX Business’ Blake Burman on Wednesday he has worn one in the past and the only reason he doesn’t wear one more often is because everyone gets tested before meeting with him. “I would. Oh, I have. I mean, people have seen me wearing one,” Trump said when Burman asked if he would wear a mask. “If I'm in a group of people where we're not, you know, 10 feet away, and -- but, usually, I'm not in that position. And everyone's tested. Because I'm the president, they get tested before they see me.” House Speaker Nancy Pelosi, D-Calif., said Sunday that a national mask mandate is “long overdue.” Democratic presidential candidate Joe Biden said last week he "would insist that everybody in public be wearing that mask.” PELOSI FAILS TO WEAR MASK PROPERLY DESPITE HER CALLS FOR NATIONAL MANDATE But Trump said he doesn’t think a national mask mandate is necessary. “I don't know if you need mandatory, because you have many places in the country where people stay very long distance. You talk about social distancing. But I'm all for masks,” Trump told Burman. https://www.foxbusiness.com/lifestyle/trump-all-for-masks-looked-like-lone-ranger-in-one CORONAVIRUS Published 20 hours ago Last Update 17 hours ago Kim Guilfoyle tests positive, Don Jr. negative, for coronavirus, Trump campaign confirms Neither traveled with President Trump aboard Air Force One, a report said By Dom Calicchio, Matt Leach | Fox News Facebook Twitter Flipboard Comments Print Email Fox News Flash top headlines for July 3 Fox News Flash top headlines are here. Check out what's clicking on Foxnews.com. Kimberly Guilfoyle, a senior adviser to the Trump 2020 Campaign and girlfriend of Donald Trump Jr., has tested positive for the coronavirus, the campaign confirmed Friday night. “After testing positive, Kimberly was immediately isolated to limit any exposure. She’s doing well, and will be retested to ensure the diagnosis is correct since she’s asymptomatic, but as a precaution will cancel all upcoming events,” Sergio Gor, chief of staff of the Trump Victory Finance Committee, told Fox News. “Donald Trump Jr. was tested negative but as a precaution is also self-isolating and is canceling all public events,” Gor added. https://www.foxnews.com/politics/kim-guilfoyle-tests-positive-don-jr-negative-for-coronavirus-trump-campaign-confirms Nascar driver Jimmie Johnson tested positive for covid19

Me too. Only reason I watch is the Bonnies since local team here. Besides that don't watch anything to NBA or college.

He still is one the best posters and a cool dude.

Good news Ya Magox. I heard lots of site online. Said covid19 because more infectious but less deadly (mutation). There is some some truth with cases and daily reports as evidence. . But who knows.Maybe why lately more cases. But IDK for sure. Just want to hear you're thoughts. Thanks man keeping good posts and updated. Enjoy seeing you here and posts. https://www.foxnews.com/health/scientists-analyzing-coronavirus-mutation-might-spread-virus-faster Coronavirus mutation may have made virus more contagious: report The mutation has been found to affect the virus' spike protein By David Aaro | Fox News Facebook Twitter Flipboard Comments Print Email Fox News Flash top headlines for June 29 Fox News Flash top headlines are here. Check out what's clicking on Foxnews.com. Scientists are trying to understand a mutation of the novel coronavirus seen around the globe that some believe could make the virus more contagious, according to a report. The mutation, officially designated D614G or "G" for short, has been found to affect the virus' spike protein, which is a structure that allows it to enter human cells. The more effective the spike protein, the easier it can enter a host's body. Research has suggested that the mutation, which changes amino acid 614 from “D” (aspartic acid) to “G” (glycine) might make the spike protein more effective, which enhances the virus’ infectiousness, according to The Washington Post. CORONAVIRUS PATIENTS EXPERIENCING 'TERRIFYING' HOSPITAL DELIRIUM: REPORTS Researchers have found that out of the roughly 50,000 genomes of the new virus uploaded to a shared database, about 70 percent carried the mutation. “The epidemiological study and our data together really explain why the [G variant’s] spread in Europe and the U.S. was really fast,” Hyeryun Choe, a virologist at Scripps Research, told the paper. “This is not just accidental.” Choe was the lead author of an unpublished study on the G variant’s enhanced infectiousness in laboratory cell cultures. He said there were a couple of reasons why "G" was more effective in spreading the virus. In the mutation, the outer parts of those proteins that bind to a human receptor were less likely to break off, which was a fault of SARS-CoV-2, the virus originated in China that causes COVID-19. CORONAVIRUS INFECTS 28 MEMBERS OF CALIFORNIA FAMILY, KILLS ONE: COVID-19 'IS A REAL THING' The faulty mechanism made it so the SARS-CoV-2 had a harder time invading host cells. He added that "G" has more spike proteins, and said those reasons made the mutation 10 times more infectious in lab experiments, according to the Post. “I think this mutation happened to compensate,” Choe said. The mutation was also found to be more contagious in four studies that have yet to be peer-reviewed. One study by scientists at the Los Alamos National Laboratory concluded that patients with the "G" mutation also have more virus in their bodies, making them more likely to spread it to others, the report said. Others believe more studies are needed to d

@BillStime Pleas answer the above

Wow total respect to that guy. Love good guys that help out the people and makes sense. Those people would love to meet and be friends with. That lady seems like a spoiled brat lol.

I think that person doesn't know. On topic happy deaths going down. If goes well next couple weeks (looking like it). Means really good going ahead.

Don't wanna knlw the hell I went thru, raped, lost might be couple kids, among many things. IDC who wants to damn know. DR treated me like a person. Living in a disabalities life sucks. Lost more then than you. Whatever people tell me here IDGAF. DR always been there I respeected him. I DON"T give a a crap about you since never give a damn crap besides you ANYWAYS,

Right agree. Warren can face me, I care about DR. I lost everything to my life to everything. I care about DR. Warren you can face me IDC. I care about DR. You don't neither, you're damn useless.

Cannot wait for DR to come back. Not many people understand me. Hell notmany people did. Drunk saying this. I love DR here. People don't wanna know been thru. Conservative's hell best friend one over certain people nowdays (LOVE CONSERATIVE). Friend on mine came over conserative made him dinner and stuff.I love DR. Kept it real. DR cannot wait for you to come back. Miss you badly man. Went thru awful crap I might be drunk cannot wait for you to come back, kept this place real. You'ree a good guy. Love Meazze post among others, they kept things real. Cannot wait to have you back man. Miss you BADLY!

CORONAVIRUS Published 4 days ago Arizona sees cases of coronavirus-linked inflammatory condition in children By Madeline Farber | Fox News Facebook Twitter Flipboard Comments Print Email Potential link between coronavirus and inflammatory syndrome in children Reaction and analysis from Dr. Roberta Debiasi, Division Chief of Pediatric Infectious Diseases at Children's National Hospital, on ‘America’s News HQ.’ As cases of the novel coronavirus continue to surge in Arizona, the state has also reported seeing a rare but serious inflammatory condition in children that experts think is likely linked to COVID-19. Banner Health in Pheonix has reported cases of multisystem inflammatory syndrome in children (MIS-C), The Arizona Republic reported, adding that cases have also been identified in Tucson. NEBRASKA SEES FIRST CASES OF RARE CORONAVIRUS-LINKED INFLAMMATORY SYNDROME IN KIDS "All of the pediatric centers around the state are on the lookout for anything that looks like this MIS-C," Dr. David Moromisato, the chief medical officer for Banner Desert and Cardon Children's medical centers, told the outlet. MIS-C is an inflammatory condition that is similar to Kawasaki disease, which causes swelling in arteries throughout the body. However, the two conditions are not the same, and MIS-C has largely been reported in children who have been infected with or exposed to COVID-19. MIS-C typically causes inflammation in the heart, lungs, kidneys, brain, skin, eyes or gastrointestinal organs. MIS-C can also cause persistent fever, rashes, vomiting and diarrhea, among other symptoms such as a red tongue and eyes. "We are seeing it from an epidemiological standpoint in areas where COVID hits hard, and then four to six weeks later we see this inflammatory syndrome show up in children," said Dr. Josh Koch, division chief of pediatric critical care medicine at Phoenix Children's Hospital. https://www.foxnews.com/health/arizona-sees-cases-of-coronavirus-linked-inflammatory-condition-in-children 4 days old like I said going to post it later. Miss you DR if reading this. Wish you were still here buddy. You're a good guy. Enjoyed you here man. Sucks got banned .

Arizona 3 days ago fox news posted on it. Busy day I'll link it later still on phone saw it couple minutes ago

Don't forget about as well CDC covid19 Kawasaki like happening to kids as well. I think school's going to use caution. On the phone hard to link and details. I think know what I mean?

Agree first part. Want a safe and effective vaccine. Understandable some people question it. Have questions as well. Until proves help people out.

Studies Report Rapid Loss of COVID-19 Antibodies The results, while preliminary, suggest that survivors of SARS-CoV-2 infection may be susceptible to reinfection within weeks or months. Amanda Heidt Jun 19, 2020 14.5K ABOVE: © ISTOCK.COM, CHRISTOPH BURGSTEDT Apair of studies published this week is shedding light on the duration of immunity following COVID-19, showing patients lose their IgG antibodies—the virus-specific, slower-forming antibodies associated with long-term immunity—within weeks or months after recovery. With COVID-19, most people who become infected do produce antibodies, and even small amounts can still neutralize the virus in vitro, according to earlier work. These latest studies could not determine if a lack of antibodies leaves people at risk of reinfection. One of the studies found that 10 percent of nearly 1,500 COVID-positive patients registered undetectable antibody levels within weeks of first showing symptoms, while the other of 74 patients found they typically lost their antibodies two to three months after recovering from the infection, especially among those who tested positive but were asymptomatic. In contrast, infections caused by coronavirus cousins such as SARS and MERS result in antibodies that remain in the body for nearly a year, according to The New York Times. The first study, published June 16 on the preprint server medRxiv, screened for antibodies in almost 1,500 coronavirus patients in Wuhan, China. The researchers compared their levels to three other groups: nearly 20,000 members of the general population; more than 1,600 patients hospitalized for reasons other than COVID-19; and more than 3,800 medical workers, whom the authors assumed had “inevitably” been exposed to the virus in its early days, meaning they should have developed antibodies. They found that while almost 90 percent of COVID-19 patients had antibodies, roughly 1 percent to 5 percent of individuals in the others groups had them as well. The authors conclude in their paper that the remaining 10 percent of infected patients with no detectable antibodies, combined with the lack of antibodies in healthcare workers, suggest that “after SARS-CoV-2 infection, people are unlikely to produce long-lasting protective antibodies against this virus.” https://www.the-scientist.com/news-opinion/studies-report-rapid-loss-of-covid-19-antibodies-67650 Cousin to this covid19 report says only a year of antibodies only last. Not for sure but really need more studies how long antibodies last. Why I don't comment antibodies until real research. Some say this covid19 only two months antibodies( who knows) Emerg Infect Dis. 2007 Oct; 13(10): 1562–1564. doi: 10.3201/eid1310.070576 PMCID: PMC2851497 PMID: 18258008 Duration of Antibody Responses after Severe Acute Respiratory Syndrome Li-Ping Wu,* Nai-Chang Wang,* Yi-Hua Chang,* Xiang-Yi Tian,* Dan-Yu Na,* Li-Yuan Zhang,* Lei Zheng,* Tao Lan,† Lin-Fa Wang,‡ and Guo-***** Liang§ Author information Copyright and License information Disclaimer This article has been cited by other articles in PMC. Go to: Abstract Among 176 patients who had had severe acute respiratory syndrome (SARS), SARS-specific antibodies were maintained for an average of 2 years, and significant reduction of immunoglobulin G–positive percentage and titers occurred in the third year. Thus, SARS patients might be susceptible to reinfection >3 years after initial exposure. Keywords: SARS, convalescent, antibodies, longitudinal study, dispatch Severe acute respiratory syndrome (SARS) represents the first pandemic transmissible disease to emerge in this century. It was caused by a previously unknown coronavirus, the SARS-associated coronavirus (SARS-CoV) (1). SARS-CoV spreads from animals to humans by a rapid adaptation and evolution process (2,3). A large number of closely related viruses are present in wildlife reservoir populations (4–6). Therefore, due to cross-species transmission of the same or a similar coronavirus, SARS could recur. Immune protection against infection with other human coronaviruses, such as OC43 and 229E, is short-lived (7). To assess SARS patients’ risk for future reinfection, we conducted a longitudinal study of immunity in convalescent patients. Go to: The Study Shanxi Province in China was 1 of the SARS epicenters during the 2002–03 outbreaks. For our study, serum samples were taken from patients in 7 designated SARS hospitals in the province during March–August 2003. Follow-up serum samples were taken at 6 months, 1, 2, and 3 years after the onset of symptoms. A total of 176 cases that met the World Health Organization (WHO) SARS case definition (8) and had known transmission history were included in this study. The study was conducted as part of a national SARS control and prevention program; use of serum from human participants was approved by the Committee for SARS Control and Prevention, Department of Science and Technology, the People’s Republic of China. Titers of serum antibodies to SARS-CoV were determined by using a commercially available ELISA kit (BJI-GBI Biotechnology, Beijing, China). The ELISA was based on an inactivated preparation of whole-virus lysate. The kit was the first commercial kit approved by the Chinese Food and Drug Administration for specific detection of SARS-CoV antibodies and has been widely used in several studies (9–11). Manufacturer’s instructions were followed without modification. Briefly, for every ELISA plate, 1 blank, 1 positive, and 2 negative controls were included. For detection of immunoglobulin G (IgG), a 1:10 dilution of testing serum (100 μL) was added to antigen-coated wells, and the plate was incubated at 37oC for 30 min. Horseradish peroxide (HRP)–conjugated antihuman IgG (100 μL) was then added for detection of bound antibodies. For detection of IgM, the incubation of primary antibodies was extended to 60 min, followed by detection with HRP-conjugated antihuman IgM. Optical density (OD) readings were deemed valid only when the negative control OD was <0.10 and the positive control was >0.50 on the same ELISA plate. The cutoff for IgG and IgM determination was defined as 0.13 and 0.11, respectively, plus OD of the negative control. When the OD of the negative control was <0.05, 0.05 was used for the calculation. In this study, the OD readings of negative controls from different testing were consistently <0.05, so the cutoff ODs for IgG and IgM were 0.18 and 0.16, respectively. Serum samples that had an OD greater than or equal to the cutoff value were considered positive. Weak positive samples (i.e., OD<2× cutoff value) were retested in duplicate on the same day; only reproducible positive results were included in the final analysis. All data were processed by using Excel version 7.0 (Microsoft Corp., Redmond, WA, USA) and SPSS software version 10 for Windows (SPSS Inc., Chicago, IL, USA). Among the cohort, 163 (92.61%) of 176 (χ2 = 200.11, p = 0.000002) were IgG positive, which indicated that most patients who met the WHO case definition were indeed infected with SARS-CoV. As shown in the Table, at ≈7 days after the onset of symptoms, the percentage who were IgG positive was ≈11.80%. This percentage continued to increase, reached 100% at 90 days, and remained largely unchanged up to 200 days. Furthermore, after 1 and 2 years 93.88% and 89.58% of patients, respectively, were IgG positive, which suggests that the immune responses were maintained in >90% of patients for 2 years. However, 3 years later, ≈50% of the convalescent population had no SARS-CoV–specific IgG. The OD changes correlated with the changes to the IgG-positive percentage, although the rate of change varied. Both the OD readings (0.93) and positive percentages peaked at 90–120 days; however, the rate of reduction of the average OD readings was much faster, dropping by 22% (0.73) and 40% (0.54) at 1 and 2 years, respectively, after symptom onset (Figure 1). Figure 1 Change of immunoglobulin G (IgG) patterns among 176 convalescent severe acute respiratory syndrome patients with known transmission history. See the Table for number of samples used for the calculation at each time point. OD, optical density. A similar observation was obtained for IgM trends in this same cohort. The percentage of patients who were IgM positive within the first 7 days was 21.4% and peaked at 76.2% after 21–30 days (Table). The patterns of IgM-positive percentage and average OD readings were similar; both peaked at 21–30 days. After 60 days, the average OD readings dropped to 0.167, close to the cutoff value of 0.160. Table Cumulative rates of SARS-CoV antibodies among 176 SARS patients with known transmission histories* Time after symptom onset, d IgG IgM† No. samples tested No. positive samples (%) Average OD No. samples tested No. positive samples (%) Average OD 0–7 17 2 (11.76) 0.046 14 3 (21.43) 0.136 8–14 26 10 (38.46) 0.190 22 14 (63.64) 0.312 15–20 22 17 (77.27) 0.351 19 12 (63.16) 0.477 21–30 36 33 (91.67) 0.493 21 16 (76.19) 0.560 31–60 72 67 (93.06) 0.627 22 14 (63.64) 0.320 61–90 35 33 (94.29) 0.745 15 5 (33.33) 0.167 91–120 11 11 (100.00) 0.965 ND ND ND 121–210 23 23 (100.00) 0.932 ND ND ND 211–365 49 46 (93.88) 0.734 ND ND ND 366–763 96 86 (89.58) 0.535 ND ND ND 764–1,265 28 15 (53.57) 0.250 ND ND ND *SARS-CoV, severe acute respiratory syndrome–associated coronavirus; Ig, immunoglobulin; OD, optical density; ND, not determined because for most samples the IgM readings already reached background level on day 90. †For some patients, we did not have enough serum to test for IgM after testing for IgG; hence, a smaller number of serum samples were tested for IgM than for IgG. Among the cohort of patients with known transmission histories, we were able to obtain a complete collection of serum samples from 18 patients at 6 months, 1, 2, and 3 years. The IgG levels of these 18 patients were analyzed separately to obtain an IgG trend that more accurately represented convalescent SARS patients (Figure 2). All 18 patients had positive IgG at 6 months and at 1 year (i.e., 100% positive); only 1 patient became IgG negative at 2 years. However, at 3 years, the positive percentage dropped to 55.56%. The reduction of OD values mimicked that of the positive percentage, again at a faster rate. The average OD readings dropped from 0.94 at 6 months to 0.64 at 1 year, which represents a reduction of 33.33%. The OD further dropped to 0.52 (45.83% reduction) by 2 years and to 0.25 by 3 years. Figure 2 Change of immunoglobulin G (IgG) patterns among 18 convalescent severe acute respiratory syndrome patients with a complete collection of sequential serum samples at the time points shown. The 18 patients were selected from the cohort of 176 patients for whom transmission history was known. OD, optical density. Go to: Conclusions To our knowledge, the 3-year follow-up conducted in this study is the longest longitudinal study ever reported. With a large number of patients who had confirmed transmission history (176) and a complete dataset for 18, the level of confidence is high that the results obtained in this study are representative for convalescent SARS patients. Similar results have been reported from longitudinal studies of SARS patients with smaller cohort size (18–98 patients) and shorter follow-up period (240 days to 2 years) (9–14). The general trend of IgM peaking at ≈1 month after symptom onset and IgG peaking at 2–4 months was consistent among different studies. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2851497/ Cousin to covid19 antibodies in the past. Real research on

Right seems scripted. Getting tired of all the madness world is bringing.

Wow on data on this.

np. In the end we all are humans. People have to support each other. Biggest downside to politics. Causes friction. You could be a amazing person. But in person don't talk much in politics. Understand people are different on it. Don't want to ruin friendship. My mother thinks biden is amazing but don't say much to her on it she's amazing respect her views. But in the end we all are humans in this together. If I was at George Floyd I'd do anything to stop the police from the person suffering. For this forward here going to respect others views try to respect them more. Let's go Buffalo bills this year as we all Bills fans lol.

Truly understand. Just looking at you're both sakes. Cooling it down to help you both out.

Jesus Fox can post here. Why PM Foxx? Sounds like you're getting aggressive. Settle damn down. If you can't handle a civil discussion then leave.